NAD⁺ Peptide

NAD⁺ Peptide is studied in experimental models for its relationship with cellular energy metabolism, redox balance, and enzymatic signaling pathways [1,2].

This peptide is for research purposes only. Not for human consumption.

Category:

Description

Cellular Energy Metabolism

NAD⁺ is central to redox reactions and energy transfer within cells [1]. Research models explore how NAD⁺-related peptides influence metabolic signaling and enzymatic activity.

Redox and Enzymatic Signaling

Studies investigate NAD⁺-associated pathways involving sirtuins, PARPs, and metabolic enzymes [2]. These pathways are evaluated through molecular and biochemical markers.

Pathway Interaction Studies

NAD⁺ Peptide is often included in comparative studies examining interactions between metabolic and stress-response signaling networks [1].

Key Specs

  • Molecule class: NAD-related peptide
  • Research context: energy metabolism, redox signaling
  • References: PubChem, PubMed [1,2]

Referenced Citations

  1. PubChem, NAD⁺: https://pubchem.ncbi.nlm.nih.gov/compound/NAD
  2. PubMed, NAD metabolism and signaling: https://pubmed.ncbi.nlm.nih.gov/25671459/

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Cellular Energy Metabolism

NAD⁺ is central to redox reactions and energy transfer within cells [1]. Research models explore how NAD⁺-related peptides influence metabolic signaling and enzymatic activity.

Redox and Enzymatic Signaling

Studies investigate NAD⁺-associated pathways involving sirtuins, PARPs, and metabolic enzymes [2]. These pathways are evaluated through molecular and biochemical markers.

Pathway Interaction Studies

NAD⁺ Peptide is often included in comparative studies examining interactions between metabolic and stress-response signaling networks [1].

Key Specs

  • Molecule class: NAD-related peptide
  • Research context: energy metabolism, redox signaling
  • References: PubChem, PubMed [1,2]

Referenced Citations

  1. PubChem, NAD⁺: https://pubchem.ncbi.nlm.nih.gov/compound/NAD
  2. PubMed, NAD metabolism and signaling: https://pubmed.ncbi.nlm.nih.gov/25671459/
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